Prof. Dr. Christine Gerber

Prof. Dr. Christine Gerber forscht schwerpunktmäßig zur Religionsgeschichte der Antike und des frühen Christentums, insbesondere zu paulinischen Briefen, Geschlechterkonstruktionen und narrativen Strategien. Ihre Arbeit verbindet theologische Textanalyse mit kulturwissenschaftlichen und geschlechtertheoretischen Perspektiven, um Machtverhältnisse und Sinnbildungsprozesse in antiken Diskursen zu untersuchen. Dabei nutzt sie Methoden der Hermeneutik, Narratologie und Diskursanalyse, um die Rezeption biblischer Texte und deren Einfluss auf soziale Transformationen zu beleuchten.

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• FOR 5323/1: Neue Schöpfung? – Aitiologische Rezeptionen der Genesiserzählung in frühchristlichen Diskursen über Kosmos, Mensch und Geschlecht. (TP 3)

  Quelle ↗

  Förderer: DFG Forschungsgruppe Zeitraum: 03/2024 - 02/2028 Projektleitung: Prof. Dr. Christine Gerber

• Resilienz in religiöser Sinngebung: Dankpsalmen und Passionserzählungen als resilienzrelevante Narrative in gesamtbiblischer Perspektive

  Quelle ↗

  Förderer: DFG Sachbeihilfe Zeitraum: 04/2025 - 03/2028 Projektleitung: Prof. Dr. Christine Gerber

• Popularphilosophie und Frühes Christentum: Texte und Anwendungen

  Quelle ↗

  Förderer: Alexander von Humboldt-Stiftung: Forschungskostenzuschuss Zeitraum: 04/2026 - 07/2026 Projektleitung: Prof. Dr. Christine Gerber

• Der erste Brief an die Thessalonicher

  2017

  Biblische Zeitschrift · DOI

• Paulus und seine ,Kinder'

  2005

  DOI

  With his Epistles, the Apostle Paul not only gave theological instruction but also cultivated individual relationships with the communities he was addressing. This study examines how the Epistles set up and secure Paul’s continuing importance for the churches if it has not already been established through his Apostolate. This is achieved above all by means of metaphors. The study focuses on the parent-child metaphors (1 Thess. 2; 1 Cor. 4; Gal. 4) with which Paul seeks to bind his “children” to himself in a special way.

• T Lymphocytes Promote the Antiviral and Inflammatory Responses of Airway Epithelial Cells

  2011

  PLoS ONE · DOI

  HYPOTHESIS: T cells modulate the antiviral and inflammatory responses of airway epithelial cells to human rhinoviruses (HRV). METHODS: Differentiated primary human nasal epithelial cells (HNEC) grown on collagen-coated filters were exposed apically to HRV14 for 6 h, washed thoroughly and co-cultured with anti-CD3/CD28 activated T cells added in the basolateral compartment for 40 h. RESULTS: HRV14 did not induce IFNγ, NOS2, CXCL8 and IL-6 in HNEC, but enhanced expression of the T cell attractant CXCL10. On the other hand, HNEC co-cultured with activated T cells produced CXCL10 at a level several orders of magnitude higher than that induced by HRV14. Albeit to a much lower degree, activated T cells also induced CXCL8, IL-6 and NOS2. Anti-IFNγ antibodies and TNF soluble receptor completely blocked CXCL10 upregulation. Furthermore, a significant correlation was observed between epithelial CXCL10 mRNA expression and the amounts of IFNγ and TNF secreted by T cells. Likewise, increasing numbers of T cells to a constant number of HNEC in co-cultures resulted in increasing epithelial CXCL10 production, attaining a plateau at high IFNγ and TNF levels. Hence, HNEC activation by T cells is induced mainly by IFNγ and/or TNF. Activated T cells also markedly inhibited viral replication in HNEC, partially through activation of the nitric oxide pathway. CONCLUSION: Cross-talk between T cells and HNEC results in activation of the latter and increases their contribution to airway inflammation and virus clearance.